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Disease

Anhedonia

Late-stage therapeutic developmentEmerging research
2
Publications
13
Clinical trials
2024
Latest publication
Latest activity
beta

Recent clinical, regulatory, research and industry developments relating to this disease.

SEP-363856 attenuates CUMS-induced depression-like behaviours and reverses hippocampal neuronal injuries.

Research2024-11-29The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry

Ketamine induced synaptic plasticity operates independently of long-term potentiation.

Research2024-06-19Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology

What's happening now

An analyst briefing on current research, clinical, regulatory and industry activity surrounding this disease.

Major developments
Clinical Milestones9View all 9
+1 more in the activity timeline below
Activity timeline9

Clinical trials

10 sponsors · 2 new · 2 completed in the last 12 months (net +2)

The current development programme across all trial phases.

Clinical programme
13
All trials
5
Active
7
Late-stage
6
Completed
Late-stage studies
Recruiting
Recently completed

Research activity

2 papers

Key research shaping understanding of this disease, combining the latest publications with the most influential evidence.

Most influential

Ketamine induced synaptic plasticity operates independently of long-term potentiation.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology · 2024 · 10 cites

SEP-363856 attenuates CUMS-induced depression-like behaviours and reverses hippocampal neuronal injuries.

The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry · 2024 · 3 cites
Recent publications

Ketamine induced synaptic plasticity operates independently of long-term potentiation.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology · 2024 · 10 cites

SEP-363856 attenuates CUMS-induced depression-like behaviours and reverses hippocampal neuronal injuries.

The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry · 2024 · 3 cites
Major themes8
  • Hippocampus2
  • Antidepressive Agents1
  • Behavior, Animal1
  • Corticosterone1
  • Depression1
  • Ketamine1
  • Long-Term Potentiation1
  • Neuronal Plasticity1
Leading journals2
  • Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology1
  • The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry1
Leading researchers8
  • Gu J1
  • Kavalali ET1
  • Li M1
  • Liu H1
  • Maes M1
  • Monteggia LM1
  • Piazza MK1
  • Xu G1
Affiliations (unnormalised)6
  • Anhui Provincial Key Laboratory for Brain Bank Construction and Resource Utilization1
  • Chaohu Hospital of Anhui Medical University1
  • Key Laboratory of Psychosomatic Medicine1
  • Sichuan Provincial Center for Mental Health1
  • The Second People's Hospital of Huizhou1
  • Vanderbilt Brain Institute1

Reference

Authoritative identity, definition & identifiers.

Defined in MeSH

Inability to experience pleasure due to impairment or dysfunction of normal psychological and neurobiological mechanisms. It is a symptom of many PSYCHOTIC DISORDERS (e.g., DEPRESSIVE DISORDER, MAJOR; and SCHIZOPHRENIA).

Identifiers
References & data sources
  • Disease identity & definition — NLM Medical Subject Headings (MeSH), public domain
  • Clinical trials — ClinicalTrials.gov (U.S. National Library of Medicine)
  • Research activity — Europe PMC (EMBL-EBI) + OpenAlex-derived paper links
  • Related entities are derived from literature co-mention (studied together) — associative, not causal.