Protein / target

Complement C3

C3P01024
UniProt: P01024
5
Research papers

Protein at a glance

Research activity

Emerging research

5 papers · latest 2024

Derived from structured UniProt, Open Targets and literature data on this page.

Research activity

5 papers · to 2024

Papers linked directly to this protein. This is the protein's own literature — descriptor-derived papers are kept separate below.

Most cited

Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy.384 cites

Wu T · Cell reports · 2019

Local externalization of phosphatidylserine mediates developmental synaptic pruning by microglia.375 cites

Scott-Hewitt N · The EMBO journal · 2020

Complement System and the Kidney: Its Role in Renal Diseases, Kidney Transplantation and Renal Cell Carcinoma.69 cites

Lasorsa F · International journal of molecular sciences · 2023

Integrity of neural extracellular matrix is required for microglia-mediated synaptic remodeling.15 cites

Cangalaya C · Glia · 2024

Induction of astrocyte reactivity promotes neurodegeneration in human pluripotent stem cell models.14 cites

Gomes C · Stem cell reports · 2024

Recent

Integrity of neural extracellular matrix is required for microglia-mediated synaptic remodeling.

Cangalaya C · Glia · 2024

Induction of astrocyte reactivity promotes neurodegeneration in human pluripotent stem cell models.

Gomes C · Stem cell reports · 2024

Complement System and the Kidney: Its Role in Renal Diseases, Kidney Transplantation and Renal Cell Carcinoma.

Lasorsa F · International journal of molecular sciences · 2023

Local externalization of phosphatidylserine mediates developmental synaptic pruning by microglia.

Scott-Hewitt N · The EMBO journal · 2020

Complement C3 Is Activated in Human AD Brain and Is Required for Neurodegeneration in Mouse Models of Amyloidosis and Tauopathy.

Wu T · Cell reports · 2019

Europe PMC papers linked directly to this protein.