Back to discover

Protein / target

Myosin-7B

Encoded byMYH7BA7E2Y1Homo sapiensSwiss-Prot
Clinically validated
Therapeutic maturity
2
Approved medicines
Open Targets target-level
View by indication →
30
Clinical trials
Small-molecule tractable
Druggability
Approved Drug

Protein at a glance

Biological role

Microfilament motor

Strongest disease association

Hypertrophic cardiomyopathy 1

Via encoding gene MYH7B · Genetic evidence · score 0.82

Therapeutic position

Established drug target

Small molecules

Derived from structured UniProt, Open Targets and literature data on this page.

Protein profile

UniProt 2026_02

Canonical identity and biological annotation from UniProt.

Function overview

Involved in muscle contraction

Subcellular location

Membrane
Domains and Gene Ontology detail (14)

Domains & features

Myosin N-terminal SH3-likeMyosin motorIQ

Gene Ontology

  • Ccardiac myofibril
  • Ccytoplasm
  • Cmembrane
  • Cmyosin filament
  • Cmyosin II complex
  • Factin filament binding
  • FATP binding
  • Fmicrofilament motor activity
  • Pregulation of CAMKK-AMPK signaling cascade
  • Pregulation of cardiac muscle cell contraction
  • Pregulation of cytosolic calcium ion concentration

1983 aa · 226 kDa · 4 isoforms

Interaction neighbourhood

STRING v12.0

Proteins with the strongest functional or physical association. Node size and line weight reflect STRING confidence; hover or select a partner to inspect one association.

MYL1PURBSOX6MYL3MED13MYL2TPM3PIGUMYLK3MYL9MYH7B

10 strongest partners — larger node and heavier line mean higher confidence

Functional and physical associations from STRING v12.0. Only associations with this protein are drawn — partner-to-partner links are not part of this evidence.

Approved medicines with mapped indications

1 medicine · 2 areas

Approved therapies targeting this protein, grouped by what they are approved to treat — the disease-first view of the medicines below. Relationships come from the canonical approved-indication graph (ChEMBL phase-4), the same source as the drug cards.

Cardiomyopathy, Hypertrophic1 medicine
Broader indication categories (1)
Cardiovascular Diseases1 medicine

Broad umbrella indications (e.g. “Neoplasms”). Shown here because every medicine also appears under a more specific disease above — kept for completeness, de-emphasised for clarity.

2 medicines meet Open Targets' target-level approved-medicine definition; the 1 shown here are those on this page with a canonical approved disease indication in the graph. Approved indications from ChEMBL (phase-4), via the canonical drug→disease graph.

Drugs targeting this protein

2

How approved and investigational drugs engage this protein — mechanism and action type, direct vs complex targeting, and how broadly each acts across other targets. A drug-first view (the section above groups the approved ones disease-first); direct binders with few recorded targets are listed first.

omecamtiv mecarbil
ApprovedActivator

Cardiac myosin activator

Acts on a complex — shared with MYH7, MYH6, MYL3 +3 more · 1 of 7 recorded protein targets

mavacamten
ApprovedInhibitor

Cardiac myosin inhibitor

Indicated for Cardiomyopathy, Hypertrophic, Cardiovascular Diseases

Acts on a complex — shared with MYH7, MYH6, MYL3 +3 more · 1 of 7 recorded protein targets

ChEMBL mechanism, action type, target identity and approved indications. Open Targets clinical status.

Translational evidence

Open Targets 26

Why this target matters therapeutically, strongest evidence first. Disease associations are gene-level (via the gene that encodes this protein) and open into the full confidence synthesis; the development universe, tractability and safety annotations are target-level, from Open Targets.

Strongest disease associations · via encoding gene MYH7B

Gene-level evidence surfaced through the gene MYH7Bthat encodes this protein — not a direct protein–disease relationship. Ranked by Forefront's causal-directness weighting, so genetically- and clinically-evidenced diseases lead over ones that merely share the literature.

Cardiomyopathy, Hypertrophic
0.86Well supported

Clinical evidence dominant · Open Targets 0.65

Hypertrophic cardiomyopathy 1
0.81Well supported

Genetic evidence dominant · Open Targets 0.50

Rare familial disorder with hypertrophic cardiomyopathy
0.81Well supported

Genetic evidence dominant · Open Targets 0.50

Left ventricular noncompaction
0.53Moderately supported

Genetic evidence dominant · Open Targets 0.40

Heart Failure
0.48Limited support

Clinical evidence dominant · Open Targets 0.39

View evidence synthesis (5)
Cardiomyopathy, HypertrophicWell supported
0.86
agreement 0.750.97
Clinical58%Genetic41%Literature1%

Open Targets aggregate 0.65 · 3 independent evidence families

Hypertrophic cardiomyopathy 1Well supported
0.81
agreement 0.690.94
Genetic100%

Open Targets aggregate 0.50 · 1 independent evidence family

Rare familial disorder with hypertrophic cardiomyopathyWell supported
0.81
agreement 0.690.94
Genetic100%

Open Targets aggregate 0.50 · 1 independent evidence family

Left ventricular noncompactionModerately supported
0.53
agreement 0.390.67
Genetic98%Literature2%

Open Targets aggregate 0.40 · 2 independent evidence families

Heart FailureLimited support
0.48
agreement 0.320.63
Clinical99%Literature1%

Open Targets aggregate 0.39 · 2 independent evidence families

The evidence agreement range shows how closely the independent evidence families agree — it is not a statistical confidence interval, and nothing here is fitted to outcome data. Derived from Open Targets evidence types under Forefront weighting; the per-type scores above show the calculation.

Show all associations
Cardiomyopathy, Hypertrophic0.65
Hypertrophic cardiomyopathy 10.50
Rare familial disorder with hypertrophic cardiomyopathy0.50
Left ventricular noncompaction0.40
Heart Failure0.39
Melanoma0.29
Ovarian dysfunction0.28

Drug development

3 compounds recorded · 2 approved · 1 in clinical development

Open Targets' development universe — every compound recorded against the target at any stage, not all approved medicines. Distinct from the 2 drugs that target this protein in Forefront's canonical graph (1 with a mapped approved indication, shown above): these count different sets and are not a subset relation.

View all recorded compounds (3)
MAVACAMTENApproval
DANICAMTIVPhase 2 3
OMECAMTIV MECARBILApproval

Open Targets known-drugs universe. Drug name and highest clinical stage only — the disease relationship is NOT read from this slice (it carries trial-context noise); approved indications come from the canonical graph above.

Tractability

Small moleculesStrong

Approved Drug support this modality.

AntibodiesEmerging

Feasibility evidence (uniprot loc high conf) — no clinical-stage drug of this modality recorded.

Protein degradersEmerging

Feasibility evidence (database ubiquitination) — no clinical-stage drug of this modality recorded.

View underlying tractability evidence (3)
SM · Approved DrugAB · UniProt loc high confPR · Database Ubiquitination

Raw Open Targets tractability assessment buckets, by modality.

Clinical trials

30

Trials of drugs that target this protein — reached indirectly through those drugs, so a trial listed here studies the drug, not the protein. Ranked by active status, then clinical phase and recency, and spread across the targeting drugs.

View all trials (26)

ClinicalTrials.gov via the drug-target graph.

What's happening now

2

Recent therapeutic activity around this target — regulatory actions, clinical trials and safety signals for 2 drugs that target this protein, plus publications where such a drug is a genuine subject. Every item is reached indirectly through the drug, not the protein itself; incidental mentions (a paper that merely measures a drug) and press items are excluded.

  1. Regulatory approval2023-06-26

    Approval: Camzyos (EMA)

    ema · regulatory · ema · via mavacamten

  2. New publication2020-11-13
    Cardiac Myosin Activation with Omecamtiv Mecarbil in Systolic Heart Failure.

    The New England journal of medicine · 2021 · 443 citations · Europe PMC · via omecamtiv mecarbil

Objective event titles are shown unmodified; the event kind and significance line are derived from structured fields. Forefront AttentionEvent stream aggregating Europe PMC Regulatory filings ClinicalTrials.gov.