Protein / target

Myosin regulatory light chain 2, ventricular/cardiac muscle isoform

MYL2P10916Homo sapiensSwiss-Prot
Clinically validated
Therapeutic maturity
2
Approved medicines
10
Clinical trials
Small-molecule tractable
Druggability
Approved Drug

Protein at a glance

Biological role

Structural constituent of muscle

Primary system

Musculoskeletal system

Strongest disease association

hypertrophic cardiomyopathy 10

Genetic evidence · score 0.92

Therapeutic maturity

Clinically validated target

2 approved medicines against this target

Druggability

Small molecule

Open Targets tractability · Approved Drug

Clinical development

2 approved · 1 in clinical development

10 linked trials

Derived from structured UniProt, Open Targets and literature data on this page.

Protein profile

UniProt 2026_02

Canonical identity and biological annotation from UniProt.

Function

Contractile protein that plays a role in heart development and function (PubMed:23365102, PubMed:32453731). Following phosphorylation, plays a role in cross-bridge cycling kinetics and cardiac muscle contraction by increasing myosin lever arm stiffness and promoting myosin head diffusion; as a consequence of the increase in maximum contraction force and calcium sensitivity of contraction force. These events altogether slow down myosin kinetics and prolong duty cycle resulting in accumulated myosins being cooperatively recruited to actin binding sites to sustain thin filament activation as a means to fine-tune myofilament calcium sensitivity to force (By similarity). During cardiogenesis plays an early role in cardiac contractility by promoting cardiac myofibril assembly (By similarity)

Subcellular location

Cytoplasm, myofibril, sarcomere, A band
Domains and Gene Ontology detail (25)

Domains & features

EF-hand 1EF-hand 2EF-hand 3

Gene Ontology

  • CA band
  • Ccardiac myofibril
  • Ccytoplasm
  • Ccytoskeleton
  • Ccytosol
  • Cmyofibril
  • Cmyosin complex
  • Csarcomere
  • Factin monomer binding
  • Fcalcium ion binding
  • Fmyosin heavy chain binding
  • Fstructural constituent of muscle

166 aa · 19 kDa

Biological roles

Reactome v97

What this protein does, drawn together from its UniProt function, Gene Ontology terms and Reactome pathways.

Muscle contractionUniProt · GO · ReactomeKinase signallingUniProt
View supporting evidence

Muscle contraction

  • ·Contractile protein that plays a role in heart development and function (PubMed:23365102…
  • ·Cytoplasm, myofibril, sarcomere, A band
  • ·myosin complex
  • ·sarcomere

Kinase signalling

  • ·Contractile protein that plays a role in heart development and function (PubMed:23365102…
View underlying pathways (1)

Concepts derived from UniProt GO Reactome — each badge above shows which sources supported that role.

Interaction neighbourhood

STRING v12.0

Proteins with the strongest functional or physical association. Node size and line weight reflect STRING confidence; hover or select a partner to inspect one association.

MYH7MYL3MYH6ACTC1ROCK2MYLK3MYLK2MYL4MYL1MYH9MYL2

10 strongest partners — larger node and heavier line mean higher confidence

Functional and physical associations from STRING v12.0. Only associations with this protein are drawn — partner-to-partner links are not part of this evidence.

Drugs targeting this protein

1

Whether each drug engages this protein directly or through a complex, and how many other targets are recorded for it. Direct binders with few recorded targets are listed first.

omecamtiv mecarbil
ApprovedActivator

Cardiac myosin activator

Appears in clinical studies involving cardiovascular disorder, heart failure, heart failure, Stroke

Acts on a complex — shared with MYH7, MYH6, MYH7B +3 more · 1 of 7 recorded protein targets

ChEMBL mechanism, action type and target identity. Open Targets clinical status and indications.

Translational evidence

Open Targets 26

Why this target matters therapeutically — disease associations, drugs in development, tractability and safety, from Open Targets.

Strongest genetic associations

Human genetic evidence — the most direct causal link between this target and a disease.

hypertrophic cardiomyopathy 100.92

Genetic · overall 0.79

hypertrophic cardiomyopathy0.87

Genetic · overall 0.78

cardiomyopathy0.85

Genetic · overall 0.52

myopathy, myofibrillar, 12, infantile-onset, with cardiomyopathy0.85

Genetic · overall 0.69

Abnormality of the cardiovascular system0.80

Genetic · overall 0.48

Highest-confidence therapeutic associations

Diseases where a drug acting on this target has already reached clinical development.

cardiovascular disorder0.76

Clinical · overall 0.52

Highest overall evidence

Remaining associations by Open Targets' aggregated evidence score.

congenital fiber-type disproportion myopathy0.50

Genetic

Rare familial disorder with hypertrophic cardiomyopathy0.50

Genetic literature

familial hypertrophic cardiomyopathy0.46

Genetic

myocardial infarction0.42

Genetic

Show all associations
hypertrophic cardiomyopathy 100.79
hypertrophic cardiomyopathy0.78
myopathy, myofibrillar, 12, infantile-onset, with cardiomyopathy0.69
cardiomyopathy0.52
cardiovascular disorder0.52
congenital fiber-type disproportion myopathy0.50
Rare familial disorder with hypertrophic cardiomyopathy0.50
Abnormality of the cardiovascular system0.48
familial hypertrophic cardiomyopathy0.46
myocardial infarction0.42

Open Targets ranks 749 associations for this target and we store the top 10 by aggregated score. The long tail is largely literature co-mention and RNA-expression evidence, not evidence that this target drives those diseases.

Known drugs · 3 total

DANICAMTIVPhase 2 3

dilated cardiomyopathy · heart failure · heart failure

OMECAMTIV MECARBILApproval

cardiovascular disorder · heart failure · heart failure

MAVACAMTENApproval

hypertrophic cardiomyopathy · cardiovascular disorder · hypertrophic cardiomyopathy

Tractability

SM · Approved DrugSM · Structure with Ligand

Clinical trials

10

Trials of drugs that target this protein — reached indirectly through those drugs, so a trial listed here studies the drug, not the protein.

ClinicalTrials.gov via the drug-target graph.

Forefront confidence

Synthesis

Our own weighting of the evidence behind each disease association. Human genetics and clinical evidence count for most; literature co-mention counts for little, because two entities sharing an abstract is not evidence that one drives the other.

hypertrophic cardiomyopathyWell supported
0.97
agreement 0.881.00
Genetic46%Clinical37%Animal model15%Literature1%Genetic literaturedup

Open Targets aggregate 0.78 · 4 independent evidence families · 1 not counted as duplicate

hypertrophic cardiomyopathy 10Well supported
0.94
agreement 0.811.00
Genetic85%Animal model15%Genetic literaturedup

Open Targets aggregate 0.79 · 2 independent evidence families · 1 not counted as duplicate

myopathy, myofibrillar, 12, infantile-onset, with cardiomyopathyWell supported
0.87
agreement 0.751.00
Genetic83%Animal model17%Genetic literaturedup

Open Targets aggregate 0.69 · 2 independent evidence families · 1 not counted as duplicate

cardiomyopathyWell supported
0.86
agreement 0.721.00
Genetic97%Literature3%

Open Targets aggregate 0.52 · 2 independent evidence families

Abnormality of the cardiovascular systemWell supported
0.80
agreement 0.680.92
Genetic100%

Open Targets aggregate 0.48 · 1 independent evidence family

This ranking differs from Open Targets' own: re-weighting moves genetically-evidenced diseases above more heavily co-mentioned ones. The evidence agreement range shows how closely the independent evidence families agree — it is not a statistical confidence interval, and nothing here is fitted to outcome data. Derived from Open Targets evidence types under Forefront weighting; the underlying per-type scores are shown above so the calculation can be checked.

What's happening now

1

Recent activity around this target, drawn from one canonical event stream. Every item is reached through a drug that targets this protein, so each event is news about that drug rather than about the protein directly.

  1. New publication2020-11-13
    Cardiac Myosin Activation with Omecamtiv Mecarbil in Systolic Heart Failure.

    The New England journal of medicine · 2021 · 443 citations · Europe PMC · via omecamtiv mecarbil

Objective event titles are shown unmodified; the event kind and significance line are derived from structured fields. Forefront AttentionEvent stream aggregating Europe PMC Regulatory filings ClinicalTrials.gov.