Protein / target

SLAM family member 7

SLAMF7Q9NQ25Homo sapiensSwiss-Prot
Clinically validated
Therapeutic maturity
1
Approved medicines
30
Clinical trials
Antibody-tractable
Druggability
Approved Drug

Protein at a glance

Biological role

Identical protein binding

Primary system

Immune system

Strongest disease association

multiple sclerosis

Genetic evidence · score 0.57

Therapeutic maturity

Clinically validated target

1 approved medicine against this target

Druggability

Antibody

Open Targets tractability · Approved Drug

Clinical development

1 approved · 1 in clinical development

30 linked trials

Derived from structured UniProt, Open Targets and literature data on this page.

Protein profile

UniProt 2026_02

Canonical identity and biological annotation from UniProt.

Function

Self-ligand receptor of the signaling lymphocytic activation molecule (SLAM) family. SLAM receptors triggered by homo- or heterotypic cell-cell interactions are modulating the activation and differentiation of a wide variety of immune cells and thus are involved in the regulation and interconnection of both innate and adaptive immune response. Activities are controlled by presence or absence of small cytoplasmic adapter proteins, SH2D1A/SAP and/or SH2D1B/EAT-2. Isoform 1 mediates NK cell activation through a SH2D1A-independent extracellular signal-regulated ERK-mediated pathway (PubMed:11698418). Positively regulates NK cell functions by a mechanism dependent on phosphorylated SH2D1B. Downstream signaling implicates PLCG1, PLCG2 and PI3K (PubMed:16339536). In addition to heterotypic NK cells-target cells interactions also homotypic interactions between NK cells may contribute to activation. However, in the absence of SH2D1B, inhibits NK cell function. Also acts inhibitory in T-cells (By similarity). May play a role in lymphocyte adhesion (PubMed:11802771). In LPS-activated monocytes negatively regulates production of pro-inflammatory cytokines (PubMed:23695528)

Subcellular location

Membrane
Domains and Gene Ontology detail (11)

Domains & features

Ig-like V-typeIg-like C2-type

Gene Ontology

  • Cexternal side of plasma membrane
  • Cplasma membrane
  • Fidentical protein binding
  • Padaptive immune response
  • Pcell adhesion
  • Pimmune response
  • Pnatural killer cell activation
  • Pnatural killer cell mediated cytotoxicity
  • PT cell activation

335 aa · 37 kDa · 7 isoforms

Biological roles

Reactome v97

What this protein does, drawn together from its UniProt function, Gene Ontology terms and Reactome pathways.

Immune signallingUniProt · GOCell adhesionGO
View supporting evidence

Immune signalling

  • ·Self-ligand receptor of the signaling lymphocytic activation molecule (SLAM) family. SLA…
  • ·adaptive immune response
  • ·immune response
  • ·T cell activation

Cell adhesion

  • ·cell adhesion
View underlying pathways (1)

Concepts derived from UniProt GO Reactome — each badge above shows which sources supported that role.

Interaction neighbourhood

STRING v12.0

Proteins with the strongest functional or physical association. Node size and line weight reflect STRING confidence; hover or select a partner to inspect one association.

CD48SH2D1BCD2SH2D1AFCGR3ACD38FCGR3BCD244KLRK1SLAMF1SLAMF7

10 strongest partners — larger node and heavier line mean higher confidence

Functional and physical associations from STRING v12.0. Only associations with this protein are drawn — partner-to-partner links are not part of this evidence.

Drugs targeting this protein

1

Whether each drug engages this protein directly or through a complex, and how many other targets are recorded for it. Direct binders with few recorded targets are listed first.

elotuzumab
Narrow target profileApprovedInhibitor

SLAM family member 7 inhibitor

Appears in clinical studies involving plasma cell myeloma, neoplasm, plasma cell myeloma, smoldering plasma cell myeloma

Direct interaction with this protein · Only this protein recorded as a target

ChEMBL mechanism, action type and target identity. Open Targets clinical status and indications.

Translational evidence

Open Targets 26

Why this target matters therapeutically — disease associations, drugs in development, tractability and safety, from Open Targets.

Strongest genetic associations

Human genetic evidence — the most direct causal link between this target and a disease.

multiple sclerosis0.57

Genetic · overall 0.35

Epstein-Barr virus infection0.53

Genetic · overall 0.32

rotator cuff syndrome0.46

Genetic · overall 0.28

Delayed puberty0.36

Genetic · overall 0.22

autism0.18

Genetic · overall 0.11

Highest-confidence therapeutic associations

Diseases where a drug acting on this target has already reached clinical development.

plasma cell myeloma0.94

Clinical · overall 0.60

neoplasm0.61

Clinical · overall 0.40

AL amyloidosis0.12

Clinical · overall 0.10

Highest overall evidence

Remaining associations by Open Targets' aggregated evidence score.

Miyoshi myopathy0.11

Literature

breast cancer0.09

Literature

Show all associations
plasma cell myeloma0.60
neoplasm0.40
multiple sclerosis0.35
Epstein-Barr virus infection0.32
rotator cuff syndrome0.28
Delayed puberty0.22
Miyoshi myopathy0.11
autism0.11
AL amyloidosis0.10
breast cancer0.09

Open Targets ranks 257 associations for this target and we store the top 10 by aggregated score. The long tail is largely literature co-mention and RNA-expression evidence, not evidence that this target drives those diseases.

Known drugs · 2 total

AZINTUXIZUMAB VEDOTINPhase 1

plasma cell myeloma

ELOTUZUMABApproval

plasma cell myeloma · neoplasm · plasma cell myeloma

Tractability

AB · Approved DrugAB · GO CC high confAB · UniProt SigP or TMHMMPR · Database UbiquitinationOC · Advanced Clinical

Clinical trials

30

Trials of drugs that target this protein — reached indirectly through those drugs, so a trial listed here studies the drug, not the protein.

Show remaining trials (24)

ClinicalTrials.gov via the drug-target graph.

Forefront confidence

Synthesis

Our own weighting of the evidence behind each disease association. Human genetics and clinical evidence count for most; literature co-mention counts for little, because two entities sharing an abstract is not evidence that one drives the other.

plasma cell myelomaModerately supported
0.74
agreement 0.590.90
Clinical84%Literature16%

Open Targets aggregate 0.60 · 2 independent evidence families

multiple sclerosisModerately supported
0.58
agreement 0.440.71
Genetic97%Literature3%

Open Targets aggregate 0.35 · 2 independent evidence families

Epstein-Barr virus infectionModerately supported
0.53
agreement 0.400.67
Genetic99%Literature1%

Open Targets aggregate 0.32 · 2 independent evidence families

neoplasmModerately supported
0.53
agreement 0.380.69
Clinical76%Literature24%

Open Targets aggregate 0.40 · 2 independent evidence families

rotator cuff syndromeLimited support
0.46
agreement 0.340.58
Genetic100%

Open Targets aggregate 0.28 · 1 independent evidence family

The evidence agreement range shows how closely the independent evidence families agree — it is not a statistical confidence interval, and nothing here is fitted to outcome data. Derived from Open Targets evidence types under Forefront weighting; the underlying per-type scores are shown above so the calculation can be checked.

What's happening now

5

Recent activity around this target, drawn from one canonical event stream. Every item is reached through a drug that targets this protein, so each event is news about that drug rather than about the protein directly.

  1. Trial status changed2026-07-30

    64407564MMY3009: A Phase 3 Randomized Study Comparing Talquetamab in Combination With Pomalidomide (Tal-P), Talquetamab in Combination With Teclistamab (Tal-Tec), and Investigator's Choice of Either Elotuzumab, Pomalidomide, and Dexamethasone (EPd) or Pom

    Status changed to Active, not recruiting · ClinicalTrials.gov · via elotuzumab

  2. New publication2023-04-07
    A Clinical and Correlative Study of Elotuzumab, Carfilzomib, Lenalidomide, and Dexamethasone (Elo-KRd) for Lenalidomide Refractory Multiple Myeloma in First Relapse.

    Clinical lymphoma, myeloma & leukemia · 2023 · 6 citations · Europe PMC · via elotuzumab

  3. New publication2018-08-10
    Targeting B Cell Maturation Antigen (BCMA) in Multiple Myeloma: Potential Uses of BCMA-Based Immunotherapy.

    Frontiers in immunology · 2018 · 218 citations · Europe PMC · via elotuzumab

  4. Accelerated approval granted2016-05-11

    Accelerated approval: Empliciti (EMA)

    ema · regulatory · ema · via elotuzumab

  5. New publication2015-06-02
    Elotuzumab Therapy for Relapsed or Refractory Multiple Myeloma.

    The New England journal of medicine · 2015 · 1,019 citations · Europe PMC · via elotuzumab

Objective event titles are shown unmodified; the event kind and significance line are derived from structured fields. Forefront AttentionEvent stream aggregating Europe PMC Regulatory filings ClinicalTrials.gov.