Protein / target

Tissue factor

F3P13726Homo sapiensSwiss-Prot
Clinically validated
Therapeutic maturity
1
Approved medicines
12
Clinical trials
Small-molecule tractable
Druggability
Structure with Ligand

Protein at a glance

Biological role

Cytokine receptor activity

Primary system

Immune system

Strongest disease association

placental abruption

Genetic evidence · score 0.49

Therapeutic maturity

Clinically validated target

1 approved medicine against this target

Druggability

Small molecule

Open Targets tractability · Structure with Ligand

Clinical development

1 approved

12 linked trials

Derived from structured UniProt, Open Targets and literature data on this page.

Protein profile

UniProt 2026_02

Canonical identity and biological annotation from UniProt.

Function

Initiates blood coagulation by forming a complex with circulating factor VII or VIIa. The [TF:VIIa] complex activates factors IX or X by specific limited proteolysis. TF plays a role in normal hemostasis by initiating the cell-surface assembly and propagation of the coagulation protease cascade

Subcellular location

MembraneSecreted
Domains and Gene Ontology detail (24)

Gene Ontology

  • Ccell surface
  • Cexternal side of plasma membrane
  • Cextracellular matrix
  • Cextracellular space
  • Cmembrane
  • Cplasma membrane
  • Cserine-type peptidase complex
  • Fcytokine receptor activity
  • Fphospholipid binding
  • Fprotease binding
  • Pactivation of blood coagulation via clotting cascade
  • Pactivation of plasma proteins involved in acute inflammatory response

295 aa · 33 kDa · 2 isoforms

Biological roles

Reactome v97

What this protein does, drawn together from its UniProt function, Gene Ontology terms and Reactome pathways.

HaemostasisUniProt · GOProteolysisUniProt · GOTranscriptional regulationGO · ReactomeImmune signallingGOCell adhesionGOApoptosis & cell deathGO
View supporting evidence

Haemostasis

  • ·Initiates blood coagulation by forming a complex with circulating factor VII or VIIa. Th…
  • ·activation of blood coagulation via clotting cascade
  • ·blood coagulation
  • ·positive regulation of platelet-derived growth factor receptor signaling pathway

Proteolysis

  • ·Initiates blood coagulation by forming a complex with circulating factor VII or VIIa. Th…
  • ·protease binding

Transcriptional regulation

  • ·positive regulation of gene expression
  • ·NGF-stimulated transcription

Immune signalling

  • ·cytokine receptor activity
  • ·activation of plasma proteins involved in acute inflammatory response
  • ·cytokine-mediated signaling pathway
  • ·positive regulation of interleukin-8 production

Cell adhesion

  • ·extracellular matrix

Apoptosis & cell death

  • ·positive regulation of endothelial cell apoptotic process
View underlying pathways (3)

Concepts derived from UniProt GO Reactome — each badge above shows which sources supported that role.

Interaction neighbourhood

STRING v12.0

Proteins with the strongest functional or physical association. Node size and line weight reflect STRING confidence; hover or select a partner to inspect one association.

F10TFPIF7F2F9THBDVWFSERPIN…F8PLGF3

10 strongest partners — larger node and heavier line mean higher confidence

Functional and physical associations from STRING v12.0. Only associations with this protein are drawn — partner-to-partner links are not part of this evidence.

Drugs targeting this protein

1

Whether each drug engages this protein directly or through a complex, and how many other targets are recorded for it. Direct binders with few recorded targets are listed first.

tisotumab vedotin
ApprovedBinding agent

Coagulation factor III binding agent

Appears in clinical studies involving cervical cancer, cervical cancer, cancer, neoplasm

Direct interaction with this protein · 1 of 16 recorded protein targets — broad pharmacology

ChEMBL mechanism, action type and target identity. Open Targets clinical status and indications.

Translational evidence

Open Targets 26

Why this target matters therapeutically — disease associations, drugs in development, tractability and safety, from Open Targets.

Strongest genetic associations

Human genetic evidence — the most direct causal link between this target and a disease.

placental abruption0.49

Genetic · overall 0.30

spontaneous coronary artery dissection0.43

Genetic · overall 0.26

macular degeneration0.33

Genetic · overall 0.20

Highest-confidence therapeutic associations

Diseases where a drug acting on this target has already reached clinical development.

cervical cancer0.91

Clinical · overall 0.56

neoplasm0.61

Clinical · overall 0.38

cancer0.61

Clinical · overall 0.38

ovarian cancer0.15

Clinical · overall 0.10

colorectal cancer0.12

Clinical · overall 0.08

Highest overall evidence

Remaining associations by Open Targets' aggregated evidence score.

retinitis pigmentosa0.08

Animal model

rheumatoid arthritis0.08

Literature

Show all associations
cervical cancer0.56
cancer0.38
neoplasm0.38
placental abruption0.30
spontaneous coronary artery dissection0.26
macular degeneration0.20
ovarian cancer0.10
retinitis pigmentosa0.08
rheumatoid arthritis0.08
colorectal cancer0.08

Open Targets ranks 1,435 associations for this target and we store the top 10 by aggregated score. The long tail is largely literature co-mention and RNA-expression evidence, not evidence that this target drives those diseases.

Known drugs · 1 total

TISOTUMAB VEDOTINApproval

cervical cancer · cervical cancer · cancer

Tractability

SM · Structure with LigandSM · High-Quality LigandSM · Druggable FamilyAB · UniProt loc high confAB · GO CC high confAB · UniProt SigP or TMHMMPR · Database UbiquitinationPR · Small Molecule BinderOC · Approved Drug

Safety liabilities

regulation of gene expression

Clinical trials

12

Trials of drugs that target this protein — reached indirectly through those drugs, so a trial listed here studies the drug, not the protein.

ClinicalTrials.gov via the drug-target graph.

Related literature

2

Papers indexed under “Thromboplastin” — a subject heading that covers this protein without being specific to it. Shown as context; not counted as this protein's own research activity.

Europe PMC literature, reached through a MeSH descriptor linked to this protein.

Forefront confidence

Synthesis

Our own weighting of the evidence behind each disease association. Human genetics and clinical evidence count for most; literature co-mention counts for little, because two entities sharing an abstract is not evidence that one drives the other.

cervical cancerModerately supported
0.69
agreement 0.530.84
Clinical96%Literature4%

Open Targets aggregate 0.56 · 2 independent evidence families

cancerLimited support
0.49
agreement 0.340.65
Clinical87%Literature13%

Open Targets aggregate 0.38 · 2 independent evidence families

placental abruptionLimited support
0.49
agreement 0.350.63
Genetic99%Literature1%

Open Targets aggregate 0.30 · 2 independent evidence families

neoplasmLimited support
0.48
agreement 0.330.64
Clinical90%Literature10%

Open Targets aggregate 0.38 · 2 independent evidence families

spontaneous coronary artery dissectionLimited support
0.43
agreement 0.290.57
Genetic98%Literature2%

Open Targets aggregate 0.26 · 2 independent evidence families

The evidence agreement range shows how closely the independent evidence families agree — it is not a statistical confidence interval, and nothing here is fitted to outcome data. Derived from Open Targets evidence types under Forefront weighting; the underlying per-type scores are shown above so the calculation can be checked.

What's happening now

3

Recent activity around this target, drawn from one canonical event stream. Every item is reached through a drug that targets this protein, so each event is news about that drug rather than about the protein directly.

  1. Regulatory approval2025-03-28

    Approval: Tivdak (EMA)

    ema · regulatory · ema · via tisotumab vedotin

  2. New publication2024-07-01
    Tisotumab Vedotin as Second- or Third-Line Therapy for Recurrent Cervical Cancer.

    The New England journal of medicine · 2024 · 107 citations · Europe PMC · via tisotumab vedotin

  3. New publication2023-08-31
    Tisotumab Vedotin in Combination With Carboplatin, Pembrolizumab, or Bevacizumab in Recurrent or Metastatic Cervical Cancer: Results From the innovaTV 205/GOG-3024/ENGOT-cx8 Study.

    Journal of clinical oncology : official journal of the American Society of Clinical Oncology · 2023 · 63 citations · Europe PMC · via tisotumab vedotin

Objective event titles are shown unmodified; the event kind and significance line are derived from structured fields. Forefront AttentionEvent stream aggregating Europe PMC Regulatory filings ClinicalTrials.gov.