Protein / target

Transthyretin

TTRP02766Homo sapiensSwiss-Prot
Clinically validated
Therapeutic maturity
12
Approved medicines
20
Clinical trials
Small-molecule tractable
Druggability
Approved Drug

Protein at a glance

Biological role

Protein-containing complex binding

Primary system

Endocrine & metabolic

Strongest disease association

amyloidosis, hereditary systemic 1

Genetic evidence · score 0.98

Therapeutic maturity

Clinically validated target

12 approved medicines against this target

Druggability

Small molecule

Open Targets tractability · Approved Drug

Clinical development

12 approved · 1 in clinical development

20 linked trials

Derived from structured UniProt, Open Targets and literature data on this page.

Protein profile

UniProt 2026_02

Canonical identity and biological annotation from UniProt.

Function

Thyroid hormone-binding protein. Probably transports thyroxine from the bloodstream to the brain

Subcellular location

SecretedCytoplasm
Domains and Gene Ontology detail (12)

Gene Ontology

  • Cazurophil granule lumen
  • Cextracellular exosome
  • Cextracellular region
  • Cextracellular space
  • Cprotein-containing complex
  • Fhormone activity
  • Fhormone binding
  • Fidentical protein binding
  • Fmolecular sequestering activity
  • Fprotein-containing complex binding
  • Ppurine nucleobase metabolic process
  • Pretinoid metabolic process

147 aa · 16 kDa

Biological roles

Reactome v97

What this protein does, drawn together from its UniProt function, Gene Ontology terms and Reactome pathways.

Metabolic enzyme activityGO
View supporting evidence

Metabolic enzyme activity

  • ·purine nucleobase metabolic process
  • ·retinoid metabolic process
View underlying pathways (6)

Concepts derived from UniProt GO Reactome — each badge above shows which sources supported that role.

Interaction neighbourhood

STRING v12.0

Proteins with the strongest functional or physical association. Node size and line weight reflect STRING confidence; hover or select a partner to inspect one association.

RBP4ALBAPOA1APPSERPIN…B2MSERPIN…APOEAPCSA2MTTR

10 strongest partners — larger node and heavier line mean higher confidence

Functional and physical associations from STRING v12.0. Only associations with this protein are drawn — partner-to-partner links are not part of this evidence.

Drugs targeting this protein

1

Whether each drug engages this protein directly or through a complex, and how many other targets are recorded for it. Direct binders with few recorded targets are listed first.

tafamidis
Narrow target profileApprovedStabiliser

Transthyretin stabiliser

Appears in clinical studies involving amyloidosis, cardiomyopathy, AL amyloidosis, Familial transthyretin-related amyloidosis

Direct interaction with this protein · Only this protein recorded as a target

ChEMBL mechanism, action type and target identity. Open Targets clinical status and indications.

Translational evidence

Open Targets 26

Why this target matters therapeutically — disease associations, drugs in development, tractability and safety, from Open Targets.

Strongest genetic associations

Human genetic evidence — the most direct causal link between this target and a disease.

amyloidosis, hereditary systemic 10.98

Genetic · overall 0.85

familial amyloid neuropathy0.95

Genetic literature · overall 0.73

Familial transthyretin-related amyloidosis0.93

Genetic literature · overall 0.59

carpal tunnel syndrome 10.83

Genetic · overall 0.72

cardiomyopathy0.81

Genetic · overall 0.73

Highest-confidence therapeutic associations

Diseases where a drug acting on this target has already reached clinical development.

amyloidosis0.95

Clinical · overall 0.71

polyneuropathy0.94

Clinical · overall 0.58

hereditary amyloidosis0.89

Clinical · overall 0.69

AL amyloidosis0.87

Clinical · overall 0.64

cardiac amyloidosis0.77

Clinical · overall 0.57

Show all associations
amyloidosis, hereditary systemic 10.85
cardiomyopathy0.73
familial amyloid neuropathy0.73
carpal tunnel syndrome 10.72
amyloidosis0.71
hereditary amyloidosis0.69
AL amyloidosis0.64
Familial transthyretin-related amyloidosis0.59
polyneuropathy0.58
cardiac amyloidosis0.57

Open Targets ranks 1,611 associations for this target and we store the top 10 by aggregated score. The long tail is largely literature co-mention and RNA-expression evidence, not evidence that this target drives those diseases.

Known drugs · 13 total

ACORAMIDIS HYDROCHLORIDEApproval

cardiac amyloidosis · cardiomyopathy · male infertility

REVUSIRANPhase 3

amyloidosis · cardiomyopathy · cardiac amyloidosis

EPLONTERSENApproval

familial amyloid neuropathy · familial amyloid neuropathy · amyloidosis

TAFAMIDISApproval

amyloidosis · cardiomyopathy · AL amyloidosis

TAFAMIDIS MEGLUMINEApproval

AL amyloidosis · amyloidosis · cardiomyopathy

VUTRISIRANApproval

hereditary amyloidosis · familial amyloid neuropathy · familial amyloid neuropathy

PATISIRAN SODIUMApproval

polyneuropathy · amyloidosis · hereditary amyloidosis

INOTERSENApproval

hereditary amyloidosis · familial amyloid neuropathy · polyneuropathy

ACORAMIDISApproval

cardiovascular disorder · familial amyloid neuropathy · amyloidosis

PATISIRANApproval

amyloidosis · hereditary amyloidosis · cardiac amyloidosis

Tractability

SM · Approved DrugSM · Structure with LigandSM · High-Quality LigandSM · High-Quality PocketSM · Druggable FamilyAB · GO CC high confAB · UniProt loc med confAB · UniProt SigP or TMHMMPR · Half-life DataPR · Small Molecule BinderOC · Approved Drug

Safety liabilities

Cognitive Function, Decreased

Clinical trials

20

Trials of drugs that target this protein — reached indirectly through those drugs, so a trial listed here studies the drug, not the protein.

Show remaining trials (14)

ClinicalTrials.gov via the drug-target graph.

Related literature

7

Papers indexed under “Prealbumin” — a subject heading that covers this protein without being specific to it. Shown as context; not counted as this protein's own research activity.

Europe PMC literature, reached through a MeSH descriptor linked to this protein.

Forefront confidence

Synthesis

Our own weighting of the evidence behind each disease association. Human genetics and clinical evidence count for most; literature co-mention counts for little, because two entities sharing an abstract is not evidence that one drives the other.

amyloidosis, hereditary systemic 1Well supported
0.99
agreement 0.881.00
Genetic72%Somatic mutation28%Genetic literaturedup

Open Targets aggregate 0.85 · 2 independent evidence families · 1 not counted as duplicate

cardiomyopathyWell supported
0.94
agreement 0.831.00
Genetic52%Clinical44%Literature3%Genetic literaturedup

Open Targets aggregate 0.73 · 3 independent evidence families · 1 not counted as duplicate

familial amyloid neuropathyWell supported
0.94
agreement 0.821.00
Genetic literature48%Clinical45%Literature7%

Open Targets aggregate 0.73 · 3 independent evidence families

amyloidosisWell supported
0.91
agreement 0.811.00
Clinical48%Genetic43%Literature9%

Open Targets aggregate 0.71 · 3 independent evidence families

hereditary amyloidosisWell supported
0.91
agreement 0.801.00
Genetic50%Clinical47%Literature3%Genetic literaturedup

Open Targets aggregate 0.69 · 3 independent evidence families · 1 not counted as duplicate

The evidence agreement range shows how closely the independent evidence families agree — it is not a statistical confidence interval, and nothing here is fitted to outcome data. Derived from Open Targets evidence types under Forefront weighting; the underlying per-type scores are shown above so the calculation can be checked.

What's happening now

3

Recent activity around this target, drawn from one canonical event stream. Every item is reached through a drug that targets this protein, so each event is news about that drug rather than about the protein directly.

  1. CHMP positive opinion2026-07-20

    CHMP positive opinion: Tafamidis Accord (EMA)

    ema · regulatory · ema · via tafamidis

  2. New publication2024-01-01
    Effect of Tafamidis on Cardiac Function in Patients With Transthyretin Amyloid Cardiomyopathy: A Post Hoc Analysis of the ATTR-ACT Randomized Clinical Trial.

    JAMA cardiology · 2024 · 56 citations · Europe PMC · via tafamidis

  3. Regulatory approval2011-11-16

    Approval: Vyndaqel (EMA)

    ema · regulatory · ema · via tafamidis

Objective event titles are shown unmodified; the event kind and significance line are derived from structured fields. Forefront AttentionEvent stream aggregating Europe PMC Regulatory filings ClinicalTrials.gov.