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Disease

Brain Injuries

Late-stage therapeutic developmentEmerging researchRising momentum
15
Publications
19
Clinical trials
5
Related conditions
1
Related proteins
2025
Latest publication
Latest activity
beta

Recent clinical, regulatory, research and industry developments relating to this disease.

Cognitive Motor Dissociation in Disorders of Consciousness.

Research2024-08-01The New England journal of medicine

Evolution of brain injury and neurological dysfunction after cardiac arrest in the rat - A multimodal and comprehensive model.

Research2024-05-21Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism

Sex Differences in Organophosphate Model of Benzodiazepine-Refractory Status Epilepticus and Neuronal Damage.

Research2024-01-17The Journal of pharmacology and experimental therapeutics

Secretome as a Tool to Treat Neurological Conditions: Are We Ready?

Research2023-11-20International journal of molecular sciences

Genomic analysis of reactive astrogliosis.

Research2012-05-01The Journal of neuroscience : the official journal of the Society for Neuroscience

What's happening now

An analyst briefing on current research, clinical, regulatory and industry activity surrounding this disease.

Executive briefingUpdating summary…Momentum: Low
Key developments
  • 2 clinical trials expected to report results, the earliest in Q3 2028.

Clinical trials

15 sponsors · 0 new · 0 completed in the last 12 months (net -1)

The current development programme across all trial phases.

Clinical programme
19
All trials
5
Active
14
Late-stage
6
Completed
Late-stage studies
Recruiting
Recently completed

Research activity

15 papers

Key research shaping understanding of this disease, combining the latest publications with the most influential evidence.

Publications over time
20122025
Most influential

Genomic analysis of reactive astrogliosis.

The Journal of neuroscience : the official journal of the Society for Neuroscience · 2012 · 1,980 cites

Interleukin-6, a major cytokine in the central nervous system.

International journal of biological sciences · 2012 · 805 cites

Cognitive Motor Dissociation in Disorders of Consciousness.

The New England journal of medicine · 2024 · 101 cites
Recent publications
Major themes8
  • Brain Injuries9
  • Biomarkers1
  • Brain Ischemia1
  • Brain Neoplasms1
  • Coma, Post-Head Injury1
  • Consciousness Disorders1
  • COVID-191
  • Dissociative Disorders1
Leading journals6
  • Nature communications2
  • Biomolecules1
  • Brain : a journal of neurology1
  • Chinese medical journal1
  • Circulation1
  • Frontiers in immunology1
Leading researchers8
  • Laureys S2
  • Menon DK2
  • Naccache L2
  • Adelson PD1
  • Albucher JF1
  • Allanson J1
  • Andelic N1
  • Araújo B1
Affiliations (unnormalised)6
  • Alder Hey Children's Hospital NHS Foundation Trust1
  • Brain and Mind Research Institute1
  • Cambridge University Hospitals NHS Foundation1
  • Center for Brain Immunology and Glia (BIG)1
  • Center for Neuroscience and Cell Biology (CNC-UC)1
  • Center for Vaccine Research and Pandemic Preparedness (C-VARPP)1

Disease biology

1 match

Key proteins & gene products studied in this disease. Number shows shared papers.

Related conditions

5 matches

Diseases frequently studied alongside this one. Number shows shared papers.

Disease profile

A grounded synthesis of the condition — overview, causes, mechanism, risk factors and current standard of care.

Overview

Brain injuries are acute or chronic injuries affecting the brain, including the cerebral hemispheres, cerebellum, and brain stem. Clinical manifestations vary with the type and location of injury, and can include diffuse injury with diffuse axonal injury or post-traumatic coma, as well as localized injury with neurobehavioral changes or focal neurologic deficits such as hemiparesis.

Causes

The grounding supports traumatic injury as a cause of brain injury, including diffuse trauma and localized injury. It also indicates that brain injury can occur as a pathological process in other neurological disorders, but does not specify additional causes or aetiologies.

Pathophysiology

The literature emphasizes secondary injury mechanisms, especially neuroinflammatory processes involving interactions between the central nervous system and the peripheral immune system. It also supports biomarker evidence of neuronal, axonal, astrocytic, and demyelinating injury, and notes altered interleukin-6 signaling, unconventional T-cell involvement, and changes in thyroid hormone availability after brain injury.

Risk factors

The supplied grounding does not identify specific risk factors for developing brain injuries. It only indicates that injury severity and clinical manifestations depend on the nature and location of the injury.

Current standard of care

The grounding does not provide a conventional treatment standard for brain injuries at the modality or drug-class level. It mentions research interest in biomarker-guided diagnosis and prognosis, immune-targeted approaches, mesenchymal stem cell secretome-based therapies, and thyroid hormone-based recovery strategies, but not established routine care.

AI-generated summary grounded in MeSH and 6 peer-reviewed sources. Informational only — not medical advice. Generated 2026-07-07.

Reference

Authoritative identity, definition & identifiers.

Defined in MeSH

Acute and chronic (see also BRAIN INJURIES, CHRONIC) injuries to the brain, including the cerebral hemispheres, CEREBELLUM, and BRAIN STEM. Clinical manifestations depend on the nature of injury. Diffuse trauma to the brain is frequently associated with DIFFUSE AXONAL INJURY or COMA, POST-TRAUMATIC. Localized injuries may be associated with NEUROBEHAVIORAL MANIFESTATIONS; HEMIPARESIS, or other focal neurologic deficits.

References & data sources
  • Disease identity & definition — NLM Medical Subject Headings (MeSH), public domain
  • Clinical trials — ClinicalTrials.gov (U.S. National Library of Medicine)
  • Research activity — Europe PMC (EMBL-EBI) + OpenAlex-derived paper links
  • Related entities are derived from literature co-mention (studied together) — associative, not causal.