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Disease

Encephalitis

Late-stage therapeutic developmentEmerging researchRising momentum
19
Publications
19
Clinical trials
11
Related conditions
5
Related proteins
2025
Latest publication
Current focus
Autoantibodies biologyTau biologyTherapeutic developmentInflammation & immunityDisease mechanisms & pathology
Latest activity
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Recent clinical, regulatory, research and industry developments relating to this disease.

Traumatic Brain Injury Causes Chronic Cortical Inflammation and Neuronal Dysfunction Mediated by Microglia.

Research2021-01-15The Journal of neuroscience : the official journal of the Society for Neuroscience

Multiple sclerosis: an immune or neurodegenerative disorder?

Research2008-01-01Annual review of neuroscience

The curry spice curcumin reduces oxidative damage and amyloid pathology in an Alzheimer transgenic mouse.

Research2001-11-01The Journal of neuroscience : the official journal of the Society for Neuroscience

What's happening now

An analyst briefing on current research, clinical, regulatory and industry activity surrounding this disease.

Clinical trials

15 sponsors · 0 new · 0 completed in the last 12 months (net +0)

The current development programme across all trial phases.

Clinical programme
19
All trials
6
Active
12
Late-stage
6
Completed
Late-stage studies
Recently completed

Research activity

19 papers

Key research shaping understanding of this disease, combining the latest publications with the most influential evidence.

Publications over time
20052025
Most influential

Neuroinflammation and M2 microglia: the good, the bad, and the inflamed.

Journal of neuroinflammation · 2014 · 1,286 cites

Multiple sclerosis: an immune or neurodegenerative disorder?

Annual review of neuroscience · 2008 · 1,237 cites

The curry spice curcumin reduces oxidative damage and amyloid pathology in an Alzheimer transgenic mouse.

The Journal of neuroscience : the official journal of the Society for Neuroscience · 2001 · 927 cites

The role of inflammation in the development of epilepsy.

Journal of neuroinflammation · 2018 · 472 cites
Recent publications
Major themes8
  • Encephalitis6
  • Hashimoto Disease3
  • Autoimmune Diseases of the Nervous System2
  • Alzheimer Disease1
  • Anti-N-Methyl-D-Aspartate Receptor Encephalitis1
  • Cognitive Dysfunction1
  • High-Throughput Nucleotide Sequencing1
  • Immune System1
Leading journals6
  • Journal of neuroinflammation5
  • The Journal of neuroscience : the official journal of the Society for Neuroscience2
  • Acta neuropathologica1
  • Alzheimer's & dementia : the journal of the Alzheimer's Association1
  • Annual review of neuroscience1
  • Brain : a journal of neurology1
Leading researchers8
  • Höftberger R2
  • Ahn J1
  • Aisen PS1
  • Alam A1
  • Askwith C1
  • Baltriukienė D1
  • Beech W1
  • Bénac N1
Affiliations (unnormalised)6
  • Medical University of Vienna2
  • University of California2
  • Alzheimer's Therapeutic Research Institute (ATRI)1
  • Beijing Fengtai Youanmen Hospital1
  • Beijing Shijitan Hospital1
  • Cardiac Electrophysiology Research and Training Center1

Disease biology

5 matches

Key proteins & gene products studied in this disease. Number shows shared papers.

Related conditions

11 matches

Diseases frequently studied alongside this one. Number shows shared papers.

Disease profile

A grounded synthesis of the condition — overview, causes, mechanism, risk factors and current standard of care.

Overview

Encephalitis is inflammation of the brain. The grounding describes it as arising from infection, autoimmune processes, toxins, and other conditions, with viral infection noted as a relatively frequent cause.

Causes

Supported causes include infection, autoimmune processes, toxins, and other conditions. Viral infections are described as a relatively frequent cause.

Pathophysiology

The literature grounding supports a neuroinflammatory process involving immune activation in the brain. It highlights cytokines, chemokines, autoantibodies, microglial activation states, and signal transduction as relevant to inflammatory injury and immune-mediated brain dysfunction.

Risk factors

The supplied grounding does not identify specific risk factors for encephalitis beyond its causes.

Current standard of care

The supplied grounding does not provide treatment guidance for encephalitis. It only supports that the condition is studied in relation to neuroinflammation and immune-mediated mechanisms, not a specific standard treatment modality or drug class.

AI-generated summary grounded in MeSH and 6 peer-reviewed sources. Informational only — not medical advice. Generated 2026-07-07.

Reference

Authoritative identity, definition & identifiers.

Defined in MeSH

Inflammation of the BRAIN due to infection, autoimmune processes, toxins, and other conditions. Viral infections (see ENCEPHALITIS, VIRAL) are a relatively frequent cause of this condition.

Identifiers
References & data sources
  • Disease identity & definition — NLM Medical Subject Headings (MeSH), public domain
  • Clinical trials — ClinicalTrials.gov (U.S. National Library of Medicine)
  • Research activity — Europe PMC (EMBL-EBI) + OpenAlex-derived paper links
  • Related entities are derived from literature co-mention (studied together) — associative, not causal.