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Disease

Alopecia

Late-stage therapeutic developmentEmerging researchSteady momentum
8
Publications
22
Clinical trials
1
Related conditions
2024
Latest publication

What's happening now

An analyst briefing on current research, clinical, regulatory and industry activity surrounding this disease.

Clinical Milestones12View all 12
+4 more in the activity timeline below
Industry & Market1View
Activity timeline13

Therapeutic landscape

Therapies with a regulatory footing for this condition, alongside the wider set of agents co-studied with it in the literature.

Approved & established therapies
Baricitinibapproved

Approval — Rheumatoid arthritis Baricitinib is indicated for the treatment of moderate to severe ac… (2017)

Clinical trials

15 sponsors · 2 new · 4 completed in the last 12 months (net +1)

The current development programme across all trial phases.

Clinical programme
22
All trials
6
Active
17
Late-stage
6
Completed
Late-stage studies
Recruiting
Recently completed

Regulatory timeline

Drug regulatory events matched to this condition by indication — EMA.

First approvals
2017emaApprovalBaricitinib· Rheumatoid arthritis Baricitinib is indicated for the treatment of moderate to severe active rheumatoid arthritis in adult patients who have responded inadequately to, or who are intolerant to one or more disease modifying anti rheumatic drugs (DMARDs). Olumiant may be used as monotherapy or in combination with methotrexate. Atopic Dermatitis Olumiant is indicated for the treatment of moderate to severe atopic dermatitis in adult and paediatric patients 2 years of age and older who are candidates for systemic therapy. Alopecia areata Baricitinib is indicated for the treatment of severe alopecia areata in adult and adolescent patients 12 years of age and older.  Juvenile idiopathic arthritis Baricitinib is indicated for the treatment of active juvenile idiopathic arthritis in patients 2 years of age and older who have had an inadequate response or intolerance to one or more prior conventional synthetic or biologic DMARDs: Polyarticular juvenile idiopathic arthritis (polyarticular rheumatoid factor positive [RF+] or negative [RF-], extended oligoarticular), Enthesitis related arthritis, and Juvenile psoriatic arthritis. Baricitinib may be used as monotherapy or in combination with methotrexate. source ↗

European Medicines Agency (CC BY 4.0). Events are matched to this condition by drug indication text — approvals/updates for drugs indicated for it, not disease-specific acts.

Research activity

8 papers

Key research shaping understanding of this disease, combining the latest publications with the most influential evidence.

Publications over time
20142024
Most influential
Recent publications
Major themes8
  • Alopecia3
  • Hair Follicle2
  • Cosmetic Techniques1
  • Esthetics1
  • Interferon-gamma1
  • Stem Cell Transplantation1
  • T-Lymphocytes, Regulatory1
  • Transgender Persons1
Leading journals6
  • Advanced materials (Deerfield Beach, Fla.)1
  • Blood1
  • Cells1
  • International journal of molecular sciences1
  • Journal of the American Academy of Dermatology1
  • Medicine1
Leading researchers8
  • Al-Tameemi W1
  • Artzi N1
  • Azzi JR1
  • Badaoui A1
  • Bajo Gauzin D1
  • Bonati LM1
  • Burke JM1
  • Cardenas-Lopez A1
Affiliations (unnormalised)6
  • American University of Beirut1
  • Brigham and Woman's Hospital1
  • Brigham and Women's Hospital1
  • Center for Infectious Medicine1
  • Center for Neuroregeneration1
  • Emory University School of Medicine1

Related conditions

1 match

Diseases frequently studied alongside this one. Number shows shared papers.

Disease profile

A grounded synthesis of the condition — overview, causes, mechanism, risk factors and current standard of care.

Overview

Alopecia is the absence of hair from areas where hair is normally present. The supplied grounding also indicates that hair loss can be discussed in specific forms such as androgenetic alopecia and chemotherapy-induced alopecia, but the core definition is simply loss of hair.

Causes

The grounding supports a genetic contribution for androgenetic alopecia, and it also identifies chemotherapy as a cause of chemotherapy-induced alopecia. Beyond these examples, no broader aetiology is supported in the supplied material.

Pathophysiology

The supplied reviews point to hair follicle biology and follicle cycling as central to hair loss, with androgenetic alopecia involving molecular and transcriptional differences between balding and non-balding scalp regions. For chemotherapy-induced alopecia, anticancer agents damage the hair follicle and alter subsequent follicle pathophysiology. Stem-cell and Wnt/growth-factor signaling are discussed as mechanisms relevant to hair regrowth and follicle development, but not as established disease mechanisms for all alopecia.

Risk factors

For androgenetic alopecia, genetics and aging are supported risk factors, and lifestyle is also mentioned as a contributing factor. No other general risk factors are supported by the supplied grounding.

Current standard of care

For chemotherapy-induced alopecia, scalp cooling is described as the only safe U.S. FDA-cleared prevention modality in the supplied review. The other grounded literature discusses regenerative and stem-cell-based approaches, platelet-rich plasma, and growth-factor or Wnt-pathway-related strategies for hair regrowth, but these are presented as investigational or emerging rather than established standard care.

AI-generated summary grounded in MeSH and 5 peer-reviewed sources. Informational only — not medical advice. Generated 2026-07-07.

Reference

Authoritative identity, definition & identifiers.

Defined in MeSH

Absence of hair from areas where it is normally present.

Identifiers
References & data sources
  • Disease identity & definition — NLM Medical Subject Headings (MeSH), public domain
  • Clinical trials — ClinicalTrials.gov (U.S. National Library of Medicine)
  • Research activity — Europe PMC (EMBL-EBI) + OpenAlex-derived paper links
  • Related entities are derived from literature co-mention (studied together) — associative, not causal.