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Disease

Glaucoma, Open-Angle

Late-stage therapeutic developmentEmerging researchCooling momentum
6
Publications
16
Clinical trials
3
Related conditions
2024
Latest publication
Latest activity
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Recent clinical, regulatory, research and industry developments relating to this disease.

What's happening now

An analyst briefing on current research, clinical, regulatory and industry activity surrounding this disease.

Therapeutic landscape

Therapies with a regulatory footing for this condition, alongside the wider set of agents co-studied with it in the literature.

Approved & established therapies
Latanoprostapproved

Approval — Catiolanze is indicated for the reduction of elevated intraocular pressure (IOP) in adult… (2023)

Netarsudilapproved

Approval — Reduction of elevated intraocular pressure (IOP) in adult patients with primary open-angl… (2019)

Travoprostapproved

Approval — Decrease of elevated intraocular pressure in adult patients with ocular hypertension or o… (2014)

Bimatoprostapproved

Approval — Reduction of elevated intraocular pressure in chronic open-angle glaucoma and ocular hype… (2002)

Clinical trials

13 sponsors · 0 new · 0 completed in the last 12 months (net +0)

The current development programme across all trial phases.

Clinical programme
16
All trials
5
Active
12
Late-stage
5
Completed
Late-stage studies
Recently completed

Regulatory timeline

Drug regulatory events matched to this condition by indication — EMA.

First approvals
2023emaApprovalLatanoprost· Catiolanze is indicated for the reduction of elevated intraocular pressure (IOP) in adult patients with open angle glaucoma or ocular hypertension. Catiolanze is indicated for the reduction of elevated IOP in children from 4 years of age and adolescents with elevated IOP and paediatric glaucoma.   source ↗
2019emaApprovalNetarsudil· Reduction of elevated intraocular pressure (IOP) in adult patients with primary open-angle glaucoma or ocular hypertension. source ↗
2014emaApprovalTravoprost· Decrease of elevated intraocular pressure in adult patients with ocular hypertension or open-angle glaucoma (see section 5.1). Decrease of elevated intraocular pressure in paediatric patients aged 3 years to < 18 years with ocular hypertension or paediatric glaucoma. source ↗
2002emaApprovalBimatoprost· Reduction of elevated intraocular pressure in chronic open-angle glaucoma and ocular hypertension (as monotherapy or as adjunctive therapy to beta-blockers). source ↗
2001emaApprovalTravoprost· Decrease of elevated intraocular pressure in adult patients with ocular hypertension or open-angle glaucoma (see section 5.1). Decrease of elevated intraocular pressure in paediatric patients aged 2 months to < 18 years with ocular hypertension or paediatric glaucoma (see section 5.1). source ↗

European Medicines Agency (CC BY 4.0). Events are matched to this condition by drug indication text — approvals/updates for drugs indicated for it, not disease-specific acts.

Research activity

6 papers

Key research shaping understanding of this disease, combining the latest publications with the most influential evidence.

Publications over time
20062024
Most influential
Recent publications
Major themes4
  • Glaucoma4
  • Glaucoma, Open-Angle4
  • Glaucoma, Angle-Closure1
  • Global Health1
Leading journals4
  • Nature communications2
  • The British journal of ophthalmology2
  • Lancet (London, England)1
  • Progress in retinal and eye research1
Leading researchers8
  • Gharahkhani P2
  • Hamel AR2
  • Khawaja AP2
  • Kolko M2
  • MacGregor S2
  • Pasquale LR2
  • Rouhana JM2
  • Segrè AV2
Affiliations (unnormalised)6
  • Harvard Medical School2
  • Icahn School of Medicine at Mount Sinai2
  • NIHR Biomedical Research Centre2
  • QIMR Berghofer Medical Research Institute2
  • Baylor College of Medicine1
  • Biomedical and Translational Informatics Institute1

Related conditions

3 matches

Diseases frequently studied alongside this one. Number shows shared papers.

Disease profile

A grounded synthesis of the condition — overview, causes, mechanism, risk factors and current standard of care.

Overview

Open-angle glaucoma is a form of glaucoma in which the anterior chamber angle remains open and the trabecular meshwork does not encroach on the base of the iris. It is a common cause of irreversible sight loss and is characterized by progressive loss of retinal ganglion cells.

Causes

The supplied grounding supports a heritable contribution to primary open-angle glaucoma, with genome-wide studies identifying many risk loci across ancestries. It also indicates that the disease is multifactorial and not completely understood, with genetic factors contributing to pathogenesis.

Pathophysiology

The literature describes glaucoma as a progressive neurodegenerative process involving retinal ganglion cell loss and optic nerve damage. Elevated intraocular pressure and/or impaired retinal blood flow may initiate injury, while oxidative stress, mitochondrial dysfunction, excitotoxicity, neuroinflammation, and trabecular meshwork dysfunction are implicated in progression and in elevated intraocular pressure.

Risk factors

Elevated intraocular pressure is the only modifiable risk factor supported by large clinical trials, although it is not always present. The disease is also associated with age-related oxidative stress and a heritable genetic predisposition, including multiple risk loci identified in genome-wide studies.

Current standard of care

Treatment is generally directed at lowering intraocular pressure. The main modalities described are topical medications, laser treatments, and surgery, including newer minimally invasive surgical approaches.

AI-generated summary grounded in MeSH and 3 peer-reviewed sources. Informational only — not medical advice. Generated 2026-07-07.

Reference

Authoritative identity, definition & identifiers.

Defined in MeSH

Glaucoma in which the angle of the anterior chamber is open and the trabecular meshwork does not encroach on the base of the iris.

References & data sources
  • Disease identity & definition — NLM Medical Subject Headings (MeSH), public domain
  • Clinical trials — ClinicalTrials.gov (U.S. National Library of Medicine)
  • Research activity — Europe PMC (EMBL-EBI) + OpenAlex-derived paper links
  • Related entities are derived from literature co-mention (studied together) — associative, not causal.