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Disease

Lupus Erythematosus, Systemic

Late-stage therapeutic developmentEmerging researchRising momentum
23
Publications
24
Clinical trials
7
Related conditions
3
Related proteins
2024
Latest publication
Current focus
Autoantibodies biologyTherapeutic developmentInflammation & immunity
Latest activity
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Recent clinical, regulatory, research and industry developments relating to this disease.

CAR-T-Cell Therapy for Systemic Lupus Erythematosus: A Comprehensive Overview.

Research2024-09-29International journal of molecular sciences

Dawn of CAR-T cell therapy in autoimmune diseases.

Research2024-04-12Chinese medical journal

Hormones and B-cell development in health and autoimmunity.

Research2024-04-12Frontiers in immunology

CD19 CAR T-Cell Therapy in Autoimmune Disease - A Case Series with Follow-up.

Research2024-02-01The New England journal of medicine

Cytokines in Systemic Lupus Erythematosus-Focus on TNF-α and IL-17.

Research2023-09-22International journal of molecular sciences

What's happening now

An analyst briefing on current research, clinical, regulatory and industry activity surrounding this disease.

Clinical Milestones11View all 11
+3 more in the activity timeline below
Activity timeline11

Therapeutic landscape

Therapies with a regulatory footing for this condition, alongside the wider set of agents co-studied with it in the literature.

Approved & established therapies
Anifrolumabapproved

Approval — Saphnelo is indicated as an add-on therapy for the treatment of adult patients with moder… (2022)

Azathioprineapproved

Approval — Jayempi is indicated in combination with other immunosuppressive agents for the prophylax… (2021)

Belimumabapproved

Approval — Benlysta is indicated as add-on therapy in patients aged 5 years and older with active, a… (2011)

Clinical trials

15 sponsors · 4 new · 0 completed in the last 12 months (net +3)

The current development programme across all trial phases.

Clinical programme
24
All trials
6
Active
18
Late-stage
6
Completed
Late-stage studies
Recruiting
Recently completed

Regulatory timeline

Drug regulatory events matched to this condition by indication — EMA.

First approvals
2022emaApprovalAnifrolumab· Saphnelo is indicated as an add-on therapy for the treatment of adult patients with moderate to severe, active autoantibody-positive systemic lupus erythematosus (SLE), despite standard therapy. source ↗
2021emaApprovalAzathioprine· Jayempi is indicated in combination with other immunosuppressive agents for the prophylaxis of transplant rejection in patients receiving allogenic kidney, liver, heart, lung or pancreas transplants. Azathioprine is indicated in immunosuppressive regimens as an adjunct to immunosuppressive agents that form the mainstay of treatment (basis immunosuppression). Jayempi is used as an immunosuppressant antimetabolite either alone or, more commonly, in combination with other agents (usually corticosteroids) and/ or procedures which influence the immune response. Jayempi is indicated in patients who are intolerant to glucocorticosteroids or if the therapeutic response is inadequate despite treatment with high doses of glucocorticosteroids, in the following diseases: severe active rheumatoid arthritis (chronic polyarthritis) that cannot be kept under control by less toxic agents (disease-modifying anti-rheumatic -medicinal products – DMARDs) auto-immune hepatitis  systemic lupus erythematosus dermatomyositis polyarteritis nodosa pemphigus vulgaris and bullous pemphigoid Behçet’s disease refractory auto-immune haemolytic anaemia, caused by warm IgG antibodies chronic refractory idiopathic thrombocytopenic purpura Jayempi is used for the treatment of moderately severe to severe forms of chronic inflammatory bowel disease (IBD) (Crohn’s disease or ulcerative colitis) in patients in whom glucocorticosteroid therapy is necessary, but where glucocorticosteroids are not tolerated, or in whom the disease is untreatable with other common means of first choice. It is also indicated in adult patients in relapsing multiple sclerosis, if an immunomodulatory therapy is indicated but beta interferon therapy is not possible, or a stable course has been achieved with previous treatment with azathioprine. 3 Jayempi is indicated for the treatment of generalised myasthenia gravis. Depending on the severity of the disease, Jayempi should be given in combination with glucocorticosteroids because of slow onset of action at the beginning of treatment and the glucocorticosteroid dose should be gradually reduced after several months of treatment. source ↗
2011emaApprovalBelimumab· Benlysta is indicated as add-on therapy in patients aged 5 years and older with active, autoantibody-positive systemic lupus erythematosus (SLE) with a high degree of disease activity (e.g., positive anti‑dsDNA and low complement) despite standard therapy. Benlysta is indicated in combination with background immunosuppressive therapies for the treatment of adult patients with active lupus nephritis. Benlysta is indicated as add-on therapy in adult patients with active, autoantibody-positive systemic lupus erythematosus (SLE) with a high degree of disease activity (e.g., positive anti dsDNA and low complement) despite standard therapy. source ↗

European Medicines Agency (CC BY 4.0). Events are matched to this condition by drug indication text — approvals/updates for drugs indicated for it, not disease-specific acts.

Research activity

23 papers

Key research shaping understanding of this disease, combining the latest publications with the most influential evidence.

Publications over time
20112024
Most influential
Recent publications
Major themes8
  • Lupus Erythematosus, Systemic15
  • Autoimmune Diseases5
  • Immunotherapy, Adoptive4
  • Receptors, Chimeric Antigen4
  • Cytokines3
  • T-Lymphocytes2
  • Anti-Infective Agents1
  • Antibodies, Monoclonal, Humanized1
Leading journals6
  • Frontiers in immunology4
  • International journal of molecular sciences3
  • Annals of the rheumatic diseases1
  • Arthritis & rheumatology (Hoboken, N.J.)1
  • Biomolecules1
  • Cellular & molecular immunology1
Leading researchers8
  • Liu R2
  • Vital EM2
  • Abdalhadi HM1
  • Abdul-Rahman T1
  • Adebusoye FT1
  • Aigner M1
  • Al-Mossawi H1
  • Alduraibi FK1
Affiliations (unnormalised)6
  • "Grigore T. Popa" University of Medicine and Pharmacy1
  • Affiliated Hospital of Southwest Medical University1
  • and the Institute of Clinical Microbiology1
  • Azienda Ospedaliero Universitaria Pisana1
  • Basic Medical College1
  • Brigham and Women's Hospital1

Disease biology

3 matches

Key proteins & gene products studied in this disease. Number shows shared papers.

Related conditions

7 matches

Diseases frequently studied alongside this one. Number shows shared papers.

Disease profile

A grounded synthesis of the condition — overview, causes, mechanism, risk factors and current standard of care.

Overview

Systemic lupus erythematosus is a chronic, relapsing, inflammatory, often febrile multisystem connective tissue disorder. It most commonly involves the skin, joints, kidneys, and serosal membranes, and is characterized by broad systemic dysfunction and the presence of LE cells in blood or bone marrow.

Causes

Its etiology is unknown. The supplied grounding describes it as an autoimmune disorder and suggests failure of normal immune regulatory mechanisms, with contributions from autoantibodies and immune dysregulation.

Pathophysiology

The disease is driven by autoimmune dysfunction with pathological autoantibody production and chronic inflammation leading to multi-organ damage. Literature grounding also points to cytokine imbalance, including TNF-α and IL-17, altered Th17/Treg balance, and neutrophil extracellular traps as contributors to inflammatory and tissue-destructive processes.

Risk factors

The supplied grounding does not support specific risk factors for systemic lupus erythematosus. It does indicate associations with vitamin D deficiency and hormonal deregulation in autoimmune disease broadly, but not as established risk factors specific to SLE.

Current standard of care

Treatment is described at the class level as antimalarial drugs, glucocorticoids, immunosuppressants, and monoclonal antibodies. The grounding also notes chimeric antigen receptor T-cell therapy as an emerging investigational approach rather than established standard care.

AI-generated summary grounded in MeSH and 6 peer-reviewed sources. Informational only — not medical advice. Generated 2026-07-07.

Reference

Authoritative identity, definition & identifiers.

Defined in MeSH

A chronic, relapsing, inflammatory, and often febrile multisystemic disorder of connective tissue, characterized principally by involvement of the skin, joints, kidneys, and serosal membranes. It is of unknown etiology, but is thought to represent a failure of the regulatory mechanisms of the autoimmune system. The disease is marked by a wide range of system dysfunctions, an elevated erythrocyte sedimentation rate, and the formation of LE cells in the blood or bone marrow.

References & data sources
  • Disease identity & definition — NLM Medical Subject Headings (MeSH), public domain
  • Clinical trials — ClinicalTrials.gov (U.S. National Library of Medicine)
  • Research activity — Europe PMC (EMBL-EBI) + OpenAlex-derived paper links
  • Related entities are derived from literature co-mention (studied together) — associative, not causal.